[86] showed that the reduction of GPx-1 activity in silenced human endothelial cells for GPx1 expression accelerated oxidative stress and increased nuclear factor kappa-light-chain-enhancer of activated B cells (NFB) and c-Jun-N-terminal kinase (JNK) activation
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Huang et al., 2021
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Transcription and activity of g-GCS is regulated by many factors and processes including GSH depletion, GSH conjugation, antioxidant levels, inflammatory cytokines, nitrosative stress, and oxidative stress (12)
The primary mechanism behind KPV anti-inflammatory action is inhibition of the NF-kB pathway
